Berita, Gosip, Gambar Artis & Selebriti Sensasi | Murai.com.my | Mylaunchpad

Berita, Gosip, Gambar Artis & Selebriti Sensasi | Murai.com.my | Mylaunchpad

Mood : Kenapa Yer?

Walaupun aku dah apply cuti tetapi hubby aku xdpt cuti,...selalunya aku akan keje..batalkan cuti tu la..tetapi hari ni aku bangun tetiba aku rasa xde semangat langsung nak g keje...rasa malas sgt..xde rasa langsung...sedangkan aku ni seorang yg lagi suka keje dari duk umah...aku pun pelik..adakah ini tanda2 awal aku akan menjadi seorang yg malas keje.....huhuhuhu...atau...masih ada rasa terkilan di hatiku ini....

masih gagal memasukkan like box facebook ke dlm blog aku ni...huhuhu

MiNDa : PTPTN

Semua org suka kalau dapat benda free macam yuran belajar free, pengangkutan awam free, macam PTPTN tu, tapi kita kenalah berpijak di bumi yang nyata..katala PTPTN dimansuhkan..pada aku yg telah meminjam wajib juga membayar dan sape yg sedang membayar kena habiskan bayar..senang citer sape yg dah pinjam kena bayar la...hutang tetap hutang..kalau xbayar berdosa tau..pergi haji pun xberkat..then kalau PTPTN dimansuhkan terus maksudnya tiada lagi PTPTN..maknanya tiada lagi bantuan pinjaman pelajaran utk anak2 akan dtg..jgnla minta kerajaan nak support 100% coz anak2 tu kan anak2 kita...kita kena berusaha memberi didikan dan pelajaran yg sempurna...kan skrg yuran sekolah dah percuma, buku teks pun dah percuma, yuran SRA pun dah percuma, bagilah masa pada kerajaan..kerajaan kita belum lagi mencapai negara maju..kita kena la ada pelaburan utk memastikan negara kita boleh survive..slowly..kita kena tolong negara kita dgn menjadi warganegara yg membayar cukai, membayar saman dan menjaga peribadi kita dan menjaga perkhidmatan yang tlh kerajaan kita berikan..Kalau dulu xde PTPTN...ramai juga yg xmampu melanjutkan pelajaran coz tersangkut di tgh jalan..bila ada PTPTN baru aku mampu smbg bljr....dan aku pun baru nak mula membayar PTPTN tu potongan gaji...coz hutang tetap hutang....itula pendapat aku...

Xjadi la 7.7..alang2 biar 10.1 terus

Bila la agaknya aku akan dapat memiliki gadget ini

MooD : Down Sikit

dah lama aku xupdate post kat blog sini...aku asyik sibuk belajar cara2 nak cantikkan blog la..asyik try ngan error...aku masih gagal nak masukkan facebook social plugins ke dalam blog aku...sampai 4 pg aku try ngan error masih gagal...hahahhaa xpe nanti aku cuba lagi...sekarang ni aku kat melaka umah mertua saja melarikan diri dari BERSHIT 3.0 tu la...lagipun lama xjenguk mak ngan ayah...jumaat lepas aku terstress la sikit coz nama aku xde dlm APC...mungkin aku yang salah..coz aku target aku boleh dapat coz aku rasa aku all out pada 2011...but aku bukan pilihan boss..nak buat camne..sedikit sebanyak isu ini telah menjatuhkan semangat aku utk bekerja pada masa sekarang....mungkin akan makan masa utk menaikkan semangat aku balik...aku pun xpasti kriteria camne yg jadi pilihan...so biarlah berlalu..tahun ni aku akan buat biasa jer...xnak lagi mengaktifkan diri..andai kata aku dapat APC tahun depan pada aku itu xde apa (just for nasib or kesian kut) coz aku tahu aku all out pada 2011 bukan 2012...

Classic polyarteritis nodosa (PAN or c-PAN) is a systemic vasculitis characterized by necrotizing inflammatory lesions that affect medium-sized and small muscular arteries, preferentially at vessel bifurcations, resulting in microaneurysm formation, aneurysmal rupture with hemorrhage, thrombosis, and, consequently, organ ischemia or infarction. Kussmaul and Maier first described PAN in 1866. The autopsy of a patient with fever, weight loss, abdominal pain, and polyneuropathy revealed areas of focal inflammatory exudations that gave rise to palpable nodules along the course of medium-sized arteries.[1] PAN, like other vasculitides, affects multiple systems and has protean manifestations, although it most commonly affects skin, joints, peripheral nerves, the gut, and the kidney.[2] The lungs are usually spared with PAN. A typical PAN patient might present with fever, night sweats, weight loss, skin ulcerations or tender nodules, and severe muscle and joint pains developing over weeks or months. (See Etiology, Clinical, and Workup.) Nonspecific, firm, tender subcutaneous nodules without livedo reticularis and/or systemic involvement may be the first sign of polyarteritis nodosa (PAN). Insight into PAN requires some understanding of how this rare disease has been defined. Periarteritis nodosa was a term used from the mid 1800s to the 1900s to describe a spectrum of systemic vasculitic disorders, including diseases that manifested as arterial aneurysms, as well as those that caused diffuse necrotizing glomerulonephritis.[3, 4] The term periarteritis nodosa was changed to polyarteritis nodosa in the mid 1900s to reflect the transmural inflammation of arteries caused by this disorder.[5] The understanding of vasculitides continued to increase by the 1980s with the discovery of antineutrophil cytoplasmic antibodies (ANCAs). Microscopic polyangiitis (MPA; formerly called microscopic polyarteritis) is an ANCA-associated systemic vasculitis that has some features similar to those of classic PAN, with the additional involvement of renal glomeruli and pulmonary capillaries. Features of PAN The American College of Rheumatology (ACR) established criteria for research purposes in order to differentiate PAN from other forms of vasculitis.[6] A committee of ACR physicians selected 10 disease features of PAN; in order for PAN to be diagnosed, at least 3 of the 10 ACR criteria should be present when radiographic or pathological diagnosis of vasculitis is made[6] (See Clinical and Workup.): Weight loss of 4 kg or more Livedo reticularis Testicular pain/tenderness Myalgia or leg weakness/tenderness Mononeuropathy or polyneuropathy Diastolic blood pressure greater than 90 mm/Hg Elevated blood urea nitrogen (BUN) or creatinine level unrelated to dehydration or obstruction Presence of hepatitis B surface antigen or antibody in serum Arteriogram demonstrating aneurysms or occlusions of the visceral arteries Biopsy of small- or medium-sized artery containing polymorphonuclear neutrophils The strong association of MPA with ANCA, as well as the pathologic and clinical differences between MPA and PAN, demonstrate that PAN and MPA are likely separate disorders. It was not until 1994 that histologic criteria to distinguish PAN from MPA were defined at the international Chapel Hill Consensus Conference (CHCC).[7] According to the CHCC criteria, the presence of vasculitis in arterioles, venules, and capillaries defines the diagnosis of MPA (although small- and medium-sized arteries may also be involved in MPA) and excludes the diagnosis of PAN. (See Clinical, Differentials, and Workup.) Stages PAN is divided into subacute, acute, and chronic stages. In the subacute stage, infiltration of mononuclear cells becomes more prominent, while in the acute stage, polymorphonuclear neutrophils infiltrate all layers of the vessel wall. (See Etiology.) In the chronic stage, fibrinoid necrosis of the vessels causes thrombosis and tissue infarction. Aneurysmal dilatations of the involved arteries, as large as 1 cm in size, are characteristic findings of PAN. Kidney lesions show predominant arteritis without glomerulonephritis; however, in patients with severe hypertension, glomerulosclerosis may be superimposed with glomerulonephritis. Pulmonary arteries are not involved, and bronchial artery involvement is uncommon. Patient education Patients should understand that PAN can be a progressive systemic disease, and further complications and the involvement of other organ systems are quite common. Many patients attempt to discontinue their medications after initial symptomatic improvement, owing to the potential for adverse effects. Therefore, the benefits of medical treatments should be discussed clearly with the patient, in addition to the risks associated with the long-term use of immunosuppressants. The use of these medications necessitates close monitoring for many years to come. (See Treatment and Medications.)

http://www.topicmuse.com/polyarteritis+nodosa

Mood : Tertanya tanya

Hari ni appointment anak aku azma haniem natasya kat hukm kul 2ptg.. Aku sampai je kat parking hukm kul 1.30, dr bo yg rawat anak aku call kata appt ni post pone next week coz ada result darah yg xkuar lg.. Tp base result yg ada mmg menunjukkan ada problem ngan darah serta salur darah anak aku.. Doc kata kalau confirm je result darah tu anak aku akan mula diberi ubat steroid utk membantunyazz dr ada sebut nama penyakitnya tapi tahula saya ni yg bekerja dgn no mana faham istilah2 dr tu.. Aku minta tlg dr smskan nama penyakit tu tp dr tu blm reply.. Aku nak tau gak la apa jenis penyakit anak aku tu.. Aku risau doc nak bg steroid kat anak aku

Mood : kebah

Ptg bawa anak2 mengeluarkan peluh kat titiwangsa..fuh penat gak.. Ramai tul org.. Demam aku pun kebah.. Tggl selsema je sikit2.. Anak ke2 aku mukhriz hakimi pg td pun dah ada tanda nak demam.. Harap2 bila dah berpeluh tu xjadila demam dia. Si adik zaquan harith yg paling non stop main... Kakak pula.. Ni yg susah hati..Syik ajak balik.. Kaki lenguh la.. Pening la.. Penat la.. Esok dia ada followe up kat hukm.. Minta2 esok dr bo tu ada la jawapan penyakit anak aku ni agar dia boleh bg ubat yg sesuai.. Bolehla kakak ni sihat cepat dan dpt sekolah seperti biasa.. Insya allah...

cybermall80: D'HERBS PREMIUM DIET PLUS FIBRE

cybermall80: D'HERBS PREMIUM DIET PLUS FIBRE

Status: Berselimut

Blk awal coz dah xtahan badan demam

Mood: rajinke aku?

Bgn pg td rasa kepala mcm dah ok... Siap2 la mai keje.. Then hujan.. Drive dlm aircond.. Kepalaku berat balik.. Hidung pun dah rasa lain mcm.. Tp kuteruskan jua..demi berkhidmat untuk negara.. Ahakkssss

Mood: Demam

Alamak.. Aku rasa kepala aku berat sangat.. Hidung mcm tersumbat.. Badan rasa seram sejuk..Ptg slm kena hujan.. Entah2 aku nak kena selsema kut..

Mood : risau

Risau lak tgk kawan opis aku yg kuar darah sikit.. Dia first pregnant .. Harap2 xde apa la

Pot luck

Masih di umah mak.. Siap pun sambal tempoyak ikan bilis.. Utk pot luck kat opis esok...Nyum nyum...

ReNuNg2KaN yer KaWaN2....

Mood: rasa nak ngumpat

Apa la nak jd ngan manusia skrg ni.. Suka menjatuhkan org lain.. Suka membawa mulut.. Jd batu api.. Jgn ingat diri tu bagus sgt.. Setakat harap rambut pacak apa barang.. Hahaha.. Xsemestinya org diam xbuat keje dan org KEPOH tu buat keje la sgt...

Sarapan dgn kawan2...

Mood : ngantuk gila...

mood : sedih

aku rasa sedih bila mengenangkan apa penyakit anak aku

susah hati aku mengenangkan apa penyakit yg kena kat anak sulungku azma haniem natasya yang ku panggil kakak..asyik cakap sakit perut sakit perut sakit perut...dah g hukm..dah buat ultrasound.. dah xray..amik darah sampai 6 botol...yg mana result nya masih belum tau....dari 22 mac lagi tak sekolah...esok xg sekolah lg la gamaknyer....semalam dah g darul syifa...ni esok nak g tempat lain lak....

dapat makan kek free...sempena ulangtahun aini couple ngan amir..hehehe....moga cepat jodoh dorang sampai...amiinnnnnnnnnnn

aku macam nak demam ngan susah hati pun yer gak...jalan2 jap la dalam opis ni..nak dijadikan citer..aku pakai la sut blazer baru aku siap ngan skirt...macam SPRM tu...hehehe...just nak rilexkan hati...huhuhuhu

tsunami.....

ni nak start belajar create blog.... tgh2 pikior...risau lak pasal gempa bumi semlm..alhamdulillah xde apa yg berlaku...
http://www.sinarharian.com.my/nasional/trauma-2004-penduduk-kuala-muda-berpindah-walaupun-tiada-arahan-1.39437